Abstracts for the BioMathDays 2008
Plenary Speakers
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Chris Bauch
TITLE
Wealth as a source of density dependence in human population growth:
evidence from the demographic transition
ABSTRACT
The phenomenon of density dependence, whereby higher population density
results in a reduced population growth rate due to resource
limitations, is a topic of intensive research in natural
populations. In modern industrialized nations, human birth rates
have been declining persistently for decades, and have now fallen below
the replacement threshold in many countries. However, unlike in
natural populations, lower birth rates in modern industrialized
countries appear to be positively correlated with resource
availability, e.g. gross domestic product (GDP) per capita. Here,
we show that declining birth rates in human populations are
actually a manifestation of density-regulated population growth brought
on by socioeconomic development, as reflected by GDP per capita.
This is demonstrated by combining empirical power law relations between
population size, GDP per capita, and fertility in a simple
theoretical model describing population dynamics in developed
countries. For a closed population, the model exhibits growth to
a globally-stable equilibrium population size, for both national
and city populations. The growth dynamics are highly sensitive to
demographic and economic power law exponents. An extended model
for a country that is open to the flow of labor, technology and capital
form other countries exhibits a good fit to long-term time series data
on population size, GDP per capita, and births rates for the United
States, France, and Japan, and provides future projections as
well. An implication of this work for sustainability is
that the earth's human population may limit itself before environmental
constraints cause widespread premature death, as predicted by
Malthusean models.
------------------------
Gerda de Vries
TITLE
Introduction to Models in Radiation Biology:
From Cell Population Models to Tumour Control Probability Curves
ABSTRACT
In this talk, I will review concepts in radiation biology that allow us
to model the effectiveness of radiation therapy in the treatment of
cancer. In particular, I will focus on the Tumour Control Probability
(TCP), which is the probability that no cancerous cells survive the
treatment. Early TCP formulae are based on simple binomial and
Poissonian statistics. They are of limited value, since they do
not take cell proliferation during the treatment period into
account. Recent TCP formulae are based on dynamic models of a
cell population, taking cell proliferation as well as the cell cycle
into account. I will conclude with a discussion of how and when
the TCP formulae are related to each other, and how they can be used to
compare the efficacy of different treatment schedules.
----------------------
Karl Peter Hadeler
TITLE
Quiescent Phases in Dynamical Systems and in Biological Modeling
ABSTRACT
In biology there are many situations where all of the dynamics or
certain components go to rest for some time and then resume activity
again and again. Examples are formation of spores, hibernation, taking
refuge from predators, etc. Such quiescent phases can affect the whole
system like in a seasonal or diurnal cycle, or individual units
(molecules, cells, animals) in a stochastic fashion. In the first case
modeling quiescence leads to time-dependent differential
equations. In
the second case we end up with what we call dynamical systems with
quiescent phases. In mathematical terms these are systems where a given
dynamics is coupled diffusely to the trivial dynamics (which
changes
nothing).
Our problem is to determine how quiescent phases change a
given
dynamics and to understand these changes in terms of a given biological
situation. We shall present some general results on reduction of growth
rates, stabilization against oscillations, and apply them to classical
examples like prey predator dynamics.
----------------------
Pauline van den Driessche
TITLE
Models for Influenza
ABSTRACT
The spread of an infectious disease, such as influenza, can be modeled
by a dynamical system that includes specific features of the disease.
The basic reproduction number R0 is an
important threshold parameter that depends on the model formulation and
parameter values related to the disease as estimated from data.
In ordinary differential equation systems, R0 can
be determined as the spectral radius of the next generation
matrix. This is illustrated for a simple influenza model, and for
a metapopulation model with individuals who travel between discrete
spatial patches. Using matrix inequalities, useful bounds on R0
are derived for this metapopulation model. A control reproduction
number is calculated for an extended influenza model that contains
vaccination and antiviral treatment. This gives insight into
quantifying strategies to control an influenza epidemic.
--------------------
Contributed Talks
----------------------
Majid Bani-Yaghoub
TITLE
Pattern Formation of Reaction-Diffusion Systems: Destabilizing
Effect of Interaction Between Signaling Pathways
ABSTRACT
Signaling pathways are known as a series of biological reactions that
affect major cellular functions (such as migration and differentiation)
by targeting specific genes. While signaling pathways regulate cellular
functions, interaction among signaling pathways (i.e. cross-talk
through endocrine, paracrine and autocrine mechanisms) may impact the
effects of signaling pathways. In the present work, interaction of
signaling pathways is considered as a perturbation to a general
reaction-diffusion (RD) system representing the chemical concentrations
in cells in a bounded domain. It is shown that, under some conditions,
weak interactions between signaling pathways may have negligible
effects on the spatial patterns generated by RD systems. This
corresponds to the topological equivalency between the perturbed and
unperturbed RD systems that may result in generation of qualitatively
the same spatial patterns. On the contrary, high levels of interaction
may result in loss of stability of Turing-type or Target patterns and
formation of new spatial patterns. Destabilizing effect of interaction
between Signaling Pathways is demonstrated by considering an
activator-inhibitor model of axon-forming potential, where the
stability of the Turing-type and Target patterns is numerically
examined.
----------------------
Aquino L. Espindola
TITLE
Probabilistic
Cellular Automata to TB spread and drug resistence emergence
ABSTRACT
The spread and evolution of bacterial resistance within human
communities is addresses in this presentation. Both, bacterial growth
dynamics within individuals and the interaction among individuals are
taken into account. We have developed a probabilistic cellular automata
model to analyse tuberculosis transition dynamics and emergence of
resistant bacteria among individuals subjected to varying levels of
antibiotic treatment. Different interaction levels among individuals
can be tested adjusting local and global contact rates. These variables
are key to determine the severity of the epidemic prevalence. We assume
that antibiotic treatment is independent of bacterial colonization.
Individuals are randomly chosen to receive treatment with antibiotics
for a given time interval. Moreover, this baseline model will allow us
to test some treatment protocols, including vaccination.
----------------------
Aquino L. Espindola
TITLE
Continuous and discrete growth models in terms of generalized
exponential functions
ABSTRACT
Here, we consider one-parameter generalizations of the logarithmic and
exponential functions, which are obtained fom the integration of
non-symmetric hyperbolas. These generalizations coincide with the ones
obtained in the context of non-extensive thermostatistics. We show that
these functions are suitable fo describe and unify a great majority of
continuous growth models. Furthermore, we also show that such a
generalization of the exponential function is also suitable to unify
most of the popular scramble and contest one-species, discrete
population dynamics models into a simple formula.
----------------------
Daihai He
TITLE
Time series analysis via mechanistic models
ABSTRACT
The purpose of time series analysis via mechanistic models is to
reconcile the known or hypothesized structure of a dynamical system
with observations collected over time. We develop a framework for
constructing nonlinear mechanistic models and carrying out inference.
Our framework permits the consideration of implicit dynamic models,
meaning statistical models for stochastic dynamical systems which are
specified by a simulation algorithm to generate sample paths. Inference
procedures that operate on implicit models are said to have the
plug-and-play property. Our work builds on recently developed
plug-and-play inference methodology for partially observed Markov
models. We introduce a class of implicitly specified Markov chains with
stochastic transition rates, and we demonstrate its applicability to
open problems in statistical inference for biological systems. As one
example, these models are shown to give a fresh perspective on measles
transmission dynamics. As a second example, we present a mechanistic
analysis of cholera incidence data, involving interaction between two
competing strains of the pathogen Vibrio cholerae.
----------------------
Olga Krylova
TITLE
Smallpox: history, virology, data analysis and more
ABSTRACT
Smallpox is one of the most devastating of human diseases. In its
12,000-year history, it has probably killed more people than any other
illness, including the plague. Interest in the study and modeling of
smallpox spread has increased in the last few years. While this
infectious disease was eradicated by the World Health Organization in
1979, it still presents a potential threat as a bio weapon. In fact,
smallpox is considered one of the two most dangerous BW agents (the
other being anthrax) because of its high case-fatality rate (>30%),
person-to-person transmission, lack of population immunity and lack of
treatment. We have analyzed weekly mortality data from the 17th, 18th
and 19th centuries in London, UK, where frequent smallpox epidemics
occurred. The data that we have cover almost 200 years and include many
smallpox epidemics. We examined the patterns in these historical data
using time series analysis. Interesting changes in smallpox dynamics
are evident over the centuries. I will discuss the history of the
disease, its virology and the results of our data analysis.
----------------------
Frithjof Lutscher
TITLE
The effect of landscape heterogeneity on spread and persistence in
integrodifference equations
ABSTRACT
The spread of non-indigenous species and diseases poses a major risk to
ecosystems and human health worldwide. The key challenges to management
and control of such invasions are to understand the conditions of
spread and the different factors influencing the speed of spread. Of
particular interest is the effect of landscape heterogeneity on the
spread of organisms. We formulate a discrete-time model for growth and
dispersal, where both of these processes vary in space. We then present
approximation formulas for the spread rate in such a heterogeneous
landscape and demonstrate their validity by comparison with numerical
simulation. We also give rules of thumb for the conditions under which
a species is able to spread in a heterogeneous landscape. We separately
consider the two cases with and without Allee effect in the population
growth function. Our results provide simple recipes for calculation of
spread rates in complex landscapes together with their limits of
validity.
----------------------
Jeff Musgrave
TITLE
An integrodifference equation model for the spread of the Emerald Ash
Borer
ABSTRACT
A biological invasion is the introduction and spread of exotic
organisms outside their native range. In this talk, the Emerald
Ash Borer, (EAB) (Agrilus planipennis),
an invasive species first discovered in Michigan in 2002, is
studied.
An important prediction of biological invasions is the spread rate at
which a population front is moving. In this talk, I present
a two-dimensional probabilistic model to predict the spread of
EAB. The model predicts the natural spread rate of EAB and the
spread rate when containment strategies are used. Results of the
model are compared with a one-dimensional integrodifference equation
model.
----------------------
Joe Tien
TITLE
Parameter estimation for bursting neural models
ABSTRACT
Parameter estimation for differential equation models is a challenging
problem. This talk will consider parameter estimation for an ODE model
of respiratory neurons that "burst". I'll discuss an approach
which uses geometric features of the differential equation to aid the
estimation, and consider possible ways that the neuronal output may be
modulated.
----------------------
Eunha Shim
TITLE
Antiviral intervention during pandemix influenza: prophylaxis and
treatment coverage levels driven by individual and societal interest
ABSTRACT
Antiviral agents will play a critical role in mitigating the next
influenza pandemic. The administration of drugs has epidemiological and
evolutionary repercussions that affect both the individual patient and
the community. An individual benefits from reduced probability and/or
severity of infection but potentially suffers adverse effects related
to antiviral drugs. From the community perspective, the positive
externality of antiviral intervention is reduced transmission, while
the negative externality is selection for drug resistance. We
develop an epidemiological game-theoretic model of pandemic influenza
in order to evaluate how the balance among these factors determines the
discrepancy between coverage levels optimized from the perspectives of
individual and societal interest. We parameterize the model with
survey data on actual perceptions regarding infection risk, the level
of resistance, the efficacy and adverse effects of antivirals, and the
willingness to pay for antivirals during pandemic influenza. We
find that the demand for antivirals driven by self-interest during
pandemic influenza would likely be far lower than those that would
maximize overall utility for the population. At current drug pricing,
this discrepancy is larger when antivirals are used for treatment than
for prophylaxis. We find that public education about the correct
information regarding the infection risk and the level of resistance
associated with antiviral drug use may be a beneficial measure to
achieve the optimal levels of antiviral drug use for population.
----------------------
Robert Smith?
TITLE
Explicitly accounting for antiretroviral drug uptake in theoretical HIV
models.
ABSTRACT
Mathematical models of HIV therapy have traditionally amalgamated the
action of antiretroviral drugs, trading the complexity of the situation
in favour of simpler - and hence mathematically tractable - models.
However, the effects of ignoring such dynamics remain underexamined. In
this paper, the traditional method of dosing (where the dose is
modelled implicitly as a proportional inhibition of viral infection and
production) is compared to a model that accounts for drug dynamics via
explicit compartments. Four limiting cases are examined: frequent
dosing of both major classes of drugs, absence of either drug, frequent
dosing of one drug alone, or frequent dosing of the other drug alone.
When both drugs are absent, both models predict that the disease-free
equilibrium will be unstable and the infection equilibrium will be
stable. When reverse transcriptase inhibitors are given frequently,
both models predict that the disease-free equilibrium will be stable
and the infection equilibrium will be unstable; this is true regardless
of whether the reverse transcriptase inhibitors act alone or in
conjunction with protease inhibitors. However, if protease inhibitors
alone are given frequently, then the implicit model predicts that the
disease-free equilibrium will be stable and the infection equilibrium
will be unstable, whereas the (more realistic) explicit model predicts
that the reverse situation may occur. It follows that the impact of
drug regimens consisting only of protease inhibitors must be urgently
re-examined.
----------------------
Naveen Vaidya
TITLE
Modeling and analysis of HIV and tuberculosis co-infection dynamics
ABSTRACT
We develop a co-epidemic model of HIV and Tuberculosis infection
dynamics. In addition to calculating the basic reproduction number, the
equilibria-analysis of the model is carried out. We present an
illustrative numerical analysis of the HIV-TB co-epidemics in India,
which we use to explore the effects of prevention and treatment
scinario. We also discuss the importance of including the effects of
each disease on the transmission and progression of the other disease.